Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000370142 | SCV000331078 | uncertain significance | not provided | 2016-05-02 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000811411 | SCV000951677 | likely pathogenic | Deficiency of acetyl-CoA acetyltransferase | 2023-02-26 | criteria provided, single submitter | clinical testing | Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt ACAT1 protein function. In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. This sequence change replaces glycine, which is neutral and non-polar, with aspartic acid, which is acidic and polar, at codon 418 of the ACAT1 protein (p.Gly418Asp). This variant is not present in population databases (gnomAD no frequency). This missense change has been observed in individuals with beta-ketothiolase deficiency (PMID: 28689740). ClinVar contains an entry for this variant (Variation ID: 281026). |
Department of Pediatrics, |
RCV000811411 | SCV000966129 | uncertain significance | Deficiency of acetyl-CoA acetyltransferase | 2019-05-05 | criteria provided, single submitter | research |