Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Color Diagnostics, |
RCV000772251 | SCV000905369 | likely benign | Hereditary cancer-predisposing syndrome | 2018-03-16 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV004569433 | SCV003223598 | likely benign | Familial adenomatous polyposis 1 | 2023-08-30 | criteria provided, single submitter | clinical testing | |
Myriad Genetics, |
RCV004569433 | SCV005084867 | benign | Familial adenomatous polyposis 1 | 2024-04-03 | criteria provided, single submitter | clinical testing | This variant is considered benign. This variant is a silent/synonymous amino acid change and it is not expected to impact splicing. |
Department of Pathology and Laboratory Medicine, |
RCV001356527 | SCV001551726 | likely benign | Carcinoma of colon | no assertion criteria provided | clinical testing | The APC p.Ser1967= variant was not identified in the literature nor was it identified in the dbSNP, LOVD 3.0 or UMD-LSDB databases. The variant was identified in ClinVar (classified as likely benign by Color). The variant was not identified in the following control databases: the Exome Aggregation Consortium (August 8th 2016) or the Genome Aggregation Database (Feb 27, 2017). The p.Ser1967= variant is not expected to have clinical significance because it does not result in a change of amino acid and is not located in a known consensus splice site. The variant occurs at a non-highly conserved nucleotide outside of the splicing consensus sequence and in silico or computational prediction software programs (SpliceSiteFinder, MaxEntScan, NNSPLICE, GeneSplicer) do not predict a difference in splicing. In summary, based on the above information, the clinical significance of this variant cannot be determined with certainty at this time although we would lean towards a more benign role for this variant. This variant is classified as likely benign. |