Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Color Diagnostics, |
RCV000581253 | SCV000687533 | likely benign | Hereditary cancer-predisposing syndrome | 2016-11-28 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000628247 | SCV000749142 | likely benign | Ataxia-telangiectasia syndrome | 2023-03-26 | criteria provided, single submitter | clinical testing | |
Gene |
RCV000842153 | SCV000984150 | likely benign | not provided | 2018-04-10 | criteria provided, single submitter | clinical testing | This variant is considered likely benign or benign based on one or more of the following criteria: it is a conservative change, it occurs at a poorly conserved position in the protein, it is predicted to be benign by multiple in silico algorithms, and/or has population frequency not consistent with disease. |
Ambry Genetics | RCV000581253 | SCV002629680 | uncertain significance | Hereditary cancer-predisposing syndrome | 2015-06-08 | criteria provided, single submitter | clinical testing | The c.4237-8T>C intronic variant results from a T to C substitution 8 nucleotides upstream from coding exon 28 in the ATM gene. This variant was not reported in population based cohorts in the following databases: Database of Single Nucleotide Polymorphisms (dbSNP), NHLBI Exome Sequencing Project (ESP), and 1000 Genomes Project. In the ESP, this variant was not observed in 6469 samples (12938 alleles) with coverage at this position. To date, this alteration has been detected with an allele frequency of approximately 0.002% (greater than 66000 alleles tested) in our clinical cohort. This nucleotide position is well conserved in available vertebrate species. Since supporting evidence is limited at this time, the clinical significance of c.4237-8T>C remains unclear. |
Myriad Genetics, |
RCV004592840 | SCV005083820 | likely benign | Familial cancer of breast | 2024-05-17 | criteria provided, single submitter | clinical testing | This variant is considered likely benign. This variant is intronic and is not expected to impact mRNA splicing. |