Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002037931 | SCV002233905 | pathogenic | Wilson disease | 2024-01-16 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Asn958Thrfs*9) in the ATP7B gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in ATP7B are known to be pathogenic (PMID: 10441329, 16283883). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with Wilson disease (PMID: 9199563, 10721669). ClinVar contains an entry for this variant (Variation ID: 1453526). For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV002037931 | SCV004216265 | pathogenic | Wilson disease | 2023-10-27 | criteria provided, single submitter | clinical testing | |
Gene |
RCV004784020 | SCV005396176 | pathogenic | not provided | 2024-05-08 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 10721669, 9199563, 32643122) |