Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV000574589 | SCV000672983 | pathogenic | Hereditary cancer-predisposing syndrome | 2017-02-17 | criteria provided, single submitter | clinical testing | The c.1500delT pathogenic mutation, located in coding exon 6 of the BLM gene, results from a deletion of one nucleotide at nucleotide position 1500, causing a translational frameshift with a predicted alternate stop codon (p.F500Lfs*2). This alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay. As such, this alteration is interpreted as a disease-causing mutation. |
Labcorp Genetics |
RCV005091419 | SCV005738341 | pathogenic | Bloom syndrome | 2024-02-04 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Phe500Leufs*2) in the BLM gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in BLM are known to be pathogenic (PMID: 17407155). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with BLM-related conditions. ClinVar contains an entry for this variant (Variation ID: 485348). For these reasons, this variant has been classified as Pathogenic. |