Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000005788 | SCV000486617 | pathogenic | Bloom syndrome | 2016-07-06 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000005788 | SCV000833195 | pathogenic | Bloom syndrome | 2023-10-13 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Ser186*) in the BLM gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in BLM are known to be pathogenic (PMID: 17407155). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with Bloom syndrome (PMID: 7585968, 17407155). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. It is commonly reported in individuals of Japanese ancestry (PMID: 17407155). ClinVar contains an entry for this variant (Variation ID: 5455). For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV000005788 | SCV004210940 | pathogenic | Bloom syndrome | 2022-03-10 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000005788 | SCV000025970 | pathogenic | Bloom syndrome | 1995-11-17 | no assertion criteria provided | literature only | |
Natera, |
RCV000005788 | SCV002089944 | pathogenic | Bloom syndrome | 2017-03-16 | no assertion criteria provided | clinical testing |