Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Evidence- |
RCV000256799 | SCV000324133 | pathogenic | Breast-ovarian cancer, familial, susceptibility to, 2 | 2016-10-18 | reviewed by expert panel | curation | Variant allele predicted to encode a truncated non-functional protein. |
Consortium of Investigators of Modifiers of BRCA1/2 |
RCV000256799 | SCV000326812 | pathogenic | Breast-ovarian cancer, familial, susceptibility to, 2 | 2015-10-02 | criteria provided, single submitter | clinical testing | |
Laboratory for Molecular Medicine, |
RCV000496292 | SCV004848416 | likely pathogenic | Hereditary breast ovarian cancer syndrome | 2020-06-19 | criteria provided, single submitter | clinical testing | The p.Ile1034AsnfsX2 variant in BRCA2 has not been reported in the literature in individuals with hereditary breast and/or ovarian cancer (HBOC) and was absent from large population studies. However, this variant was classified as pathogenic on April 22, 2016 by the ClinGen-approved ENIGMA expert panel (ClinVar SCV000324133.1). This variant is predicted to cause a frameshift, which alters the protein’s amino acid sequence beginning at position 1034 and leads to a premature termination codon 2 amino acids downstream. This alteration is then predicted to lead to a truncated or absent protein. Loss of function of the BRCA2 gene is an established disease mechanism in autosomal dominant HBOC. In summary, although additional studies are required to fully establish its clinical significance, this variant meets criteria to be classified as likely pathogenic for autosomal dominant hereditary breast and ovarian cancer. ACMG/AMP codes applied: PVS1, PM2. |
Research Molecular Genetics Laboratory, |
RCV000496292 | SCV000587655 | pathogenic | Hereditary breast ovarian cancer syndrome | 2014-01-31 | no assertion criteria provided | research |