Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000816209 | SCV000956706 | pathogenic | X-linked agammaglobulinemia with growth hormone deficiency | 2023-11-27 | criteria provided, single submitter | clinical testing | This sequence change replaces arginine, which is basic and polar, with tryptophan, which is neutral and slightly polar, at codon 562 of the BTK protein (p.Arg562Trp). This variant is not present in population databases (gnomAD no frequency). This missense change has been observed in individuals with X-linked agammaglobulinemia (PMID: 7880320, 11742281, 16951917, 17765309, 19904586). It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 11383). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt BTK protein function with a positive predictive value of 80%. For these reasons, this variant has been classified as Pathogenic. |
OMIM | RCV000012136 | SCV000032370 | pathogenic | X-linked agammaglobulinemia | 1994-10-01 | no assertion criteria provided | literature only | |
Clinical Molecular Genetics Laboratory, |
RCV000581337 | SCV000692198 | pathogenic | Autosomal recessive agammaglobulinemia 1 | 2011-08-31 | no assertion criteria provided | clinical testing |