Total submissions: 7
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000285354 | SCV000336893 | uncertain significance | not provided | 2015-11-12 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV000765367 | SCV000896632 | uncertain significance | Congenital myasthenic syndrome 4A; Congenital myasthenic syndrome 4C; Congenital myasthenic syndrome 4B | 2018-10-31 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000805415 | SCV000945370 | uncertain significance | Congenital myasthenic syndrome 4A | 2022-09-30 | criteria provided, single submitter | clinical testing | This sequence change replaces glutamic acid, which is acidic and polar, with lysine, which is basic and polar, at codon 348 of the CHRNE protein (p.Glu348Lys). This variant is present in population databases (rs757968612, gnomAD 0.02%). This variant has not been reported in the literature in individuals affected with CHRNE-related conditions. ClinVar contains an entry for this variant (Variation ID: 284315). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is not expected to disrupt CHRNE protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Revvity Omics, |
RCV000285354 | SCV003830603 | uncertain significance | not provided | 2019-05-31 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV003343740 | SCV004060532 | uncertain significance | Inborn genetic diseases | 2023-08-02 | criteria provided, single submitter | clinical testing | The c.1042G>A (p.E348K) alteration is located in exon 10 (coding exon 10) of the CHRNE gene. This alteration results from a G to A substitution at nucleotide position 1042, causing the glutamic acid (E) at amino acid position 348 to be replaced by a lysine (K). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Natera, |
RCV001833333 | SCV002093395 | uncertain significance | Congenital myasthenic syndrome | 2020-02-26 | no assertion criteria provided | clinical testing | |
Prevention |
RCV004737413 | SCV005341712 | uncertain significance | CHRNE-related disorder | 2024-09-04 | no assertion criteria provided | clinical testing | The CHRNE c.1042G>A variant is predicted to result in the amino acid substitution p.Glu348Lys. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.018% of alleles in individuals of European (non-Finnish) descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |