Total submissions: 8
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Mendelics | RCV000987988 | SCV001137530 | benign | Congenital myotonia, autosomal recessive form | 2019-05-28 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001519256 | SCV001728089 | benign | Congenital myotonia, autosomal recessive form; Congenital myotonia, autosomal dominant form | 2024-02-01 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV001579208 | SCV001806660 | benign | Congenital myotonia, autosomal dominant form | 2021-07-22 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV000987988 | SCV001806661 | benign | Congenital myotonia, autosomal recessive form | 2021-07-22 | criteria provided, single submitter | clinical testing | |
Department Of Human Genetics, |
RCV001519256 | SCV005038734 | likely benign | Congenital myotonia, autosomal recessive form; Congenital myotonia, autosomal dominant form | criteria provided, single submitter | research | Almost all patients from our cohort were homozygous for this variant c.352G>T (p.(Gly118Trp)), indicating that T represents a wild-type allele. We identified only one patient homozygous for allele G, who, however, was homozygous also for the Likely Pathogenic variant c.959A>C (p.(Ala320Val)). Only according to the publication PMID: 23152584, c.352G>T variant can have a weak effect. | |
Breakthrough Genomics, |
RCV004712951 | SCV005270762 | benign | not provided | criteria provided, single submitter | not provided | ||
Diagnostic Laboratory, |
RCV001528988 | SCV001741680 | benign | not specified | no assertion criteria provided | clinical testing | ||
Genome Diagnostics Laboratory, |
RCV001528988 | SCV001930344 | benign | not specified | no assertion criteria provided | clinical testing |