Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001931651 | SCV002203117 | pathogenic | Osteogenesis imperfecta type I | 2024-11-06 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Pro556Leufs*24) in the COL1A1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in COL1A1 are known to be pathogenic (PMID: 7942841, 9295084, 9443882). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with osteogenesis imperfecta types I and IV (PMID: 26627451, 28725987). ClinVar contains an entry for this variant (Variation ID: 1431093). For these reasons, this variant has been classified as Pathogenic. |
Laboratory of Medical Genetics, |
RCV001931651 | SCV002577543 | pathogenic | Osteogenesis imperfecta type I | 2021-01-13 | criteria provided, single submitter | clinical testing | PVS1, PM2, PP3 |
Fulgent Genetics, |
RCV002503654 | SCV002813597 | pathogenic | Osteogenesis imperfecta with normal sclerae, dominant form; Osteogenesis imperfecta, perinatal lethal; Osteogenesis imperfecta type III; Infantile cortical hyperostosis; Ehlers-Danlos syndrome, arthrochalasia type; Osteogenesis imperfecta type I; Osteoporosis; Combined osteogenesis imperfecta and Ehlers-Danlos syndrome 1 | 2022-03-16 | criteria provided, single submitter | clinical testing |