Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002241567 | SCV001420989 | pathogenic | Ehlers-Danlos syndrome, classic type, 1 | 2024-12-30 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg66*) in the COL5A1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in COL5A1 are known to be pathogenic (PMID: 23587214). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with clinical features of Ehlers-Danlos syndrome (PMID: 28485813). ClinVar contains an entry for this variant (Variation ID: 971716). For these reasons, this variant has been classified as Pathogenic. |
Laboratorio de Genetica e Diagnostico Molecular, |
RCV002241567 | SCV002512496 | pathogenic | Ehlers-Danlos syndrome, classic type, 1 | 2021-06-28 | criteria provided, single submitter | clinical testing | ACMG classification criteria: PVS1 very strong, PS4 supporting, PM2 moderate |
Fulgent Genetics, |
RCV002499430 | SCV002811688 | likely pathogenic | Ehlers-Danlos syndrome, classic type, 1; Fibromuscular dysplasia, multifocal | 2022-03-04 | criteria provided, single submitter | clinical testing |