Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000200293 | SCV000249815 | uncertain significance | not provided | 2023-11-20 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Identified independently and in conjunction with additional variants in individuals referred for genetic testing at GeneDx; segregation data is limited at this time; Has not been previously published as pathogenic or benign to our knowledge; This variant is associated with the following publications: (PMID: 22696272) |
Illumina Laboratory Services, |
RCV000341052 | SCV000478556 | uncertain significance | Ehlers-Danlos syndrome type 7A | 2016-06-14 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002229047 | SCV000631489 | benign | Ehlers-Danlos syndrome, classic type, 1 | 2023-07-29 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV003165443 | SCV003913573 | uncertain significance | Familial thoracic aortic aneurysm and aortic dissection | 2022-12-08 | criteria provided, single submitter | clinical testing | The p.P1083S variant (also known as c.3247C>T), located in coding exon 41 of the COL5A1 gene, results from a C to T substitution at nucleotide position 3247. The proline at codon 1083 is replaced by serine, an amino acid with similar properties. This amino acid position is highly conserved in available vertebrate species. In addition, the in silico prediction for this alteration is inconclusive. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Prevention |
RCV004748647 | SCV005366965 | uncertain significance | COL5A1-related disorder | 2024-03-25 | no assertion criteria provided | clinical testing | The COL5A1 c.3247C>T variant is predicted to result in the amino acid substitution p.Pro1083Ser. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0062% of alleles in individuals of African descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |