Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000200017 | SCV000249852 | uncertain significance | not provided | 2023-12-12 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; Not observed at significant frequency in large population cohorts (gnomAD); Not located in the triple helical region, where the majority of pathogenic missense variants occur (PMID: 22696272, HGMD); In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 22696272) |
Labcorp Genetics |
RCV002515350 | SCV003514025 | likely benign | Ehlers-Danlos syndrome, classic type, 1 | 2023-10-22 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV003165445 | SCV003857691 | uncertain significance | Familial thoracic aortic aneurysm and aortic dissection | 2022-12-15 | criteria provided, single submitter | clinical testing | The p.C1645Y variant (also known as c.4934G>A), located in coding exon 62 of the COL5A1 gene, results from a G to A substitution at nucleotide position 4934. The cysteine at codon 1645 is replaced by tyrosine, an amino acid with highly dissimilar properties. This amino acid position is well conserved in available vertebrate species. In addition, the in silico prediction for this alteration is inconclusive. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV004700581 | SCV005204972 | uncertain significance | not specified | 2024-06-09 | criteria provided, single submitter | clinical testing |