Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV003065803 | SCV003458265 | likely benign | not provided | 2024-01-03 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV003250734 | SCV003950301 | uncertain significance | Inborn genetic diseases | 2023-06-06 | criteria provided, single submitter | clinical testing | The c.3364C>T (p.R1122C) alteration is located in exon 25 (coding exon 25) of the COL7A1 gene. This alteration results from a C to T substitution at nucleotide position 3364, causing the arginine (R) at amino acid position 1122 to be replaced by a cysteine (C). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Prevention |
RCV003420317 | SCV004116096 | uncertain significance | COL7A1-related disorder | 2023-04-07 | criteria provided, single submitter | clinical testing | The COL7A1 c.3364C>T variant is predicted to result in the amino acid substitution p.Arg1122Cys. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0080% of alleles in individuals of African descent in gnomAD (http://gnomad.broadinstitute.org/variant/3-48624035-G-A). At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |