Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001210402 | SCV001381887 | pathogenic | not provided | 2020-04-15 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Loss-of-function variants in COL7A1 are known to be pathogenic (PMID: 16971478). This variant has been observed in individuals affected with epidermolysis bullosa (PMID: 10944088, 16971478). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the ExAC database. This sequence change creates a premature translational stop signal (p.Gly2233Argfs*57) in the COL7A1 gene. It is expected to result in an absent or disrupted protein product. |
Center for Genomic Medicine, |
RCV001210402 | SCV004024916 | pathogenic | not provided | 2025-03-04 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001210402 | SCV004168000 | pathogenic | not provided | 2023-11-15 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 21448560, 10944088, 16971478) |