Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000666078 | SCV000790317 | likely pathogenic | Carnitine palmitoyl transferase II deficiency, severe infantile form | 2017-03-20 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001861748 | SCV002125123 | pathogenic | Carnitine palmitoyltransferase II deficiency | 2023-11-20 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Thr224Leufs*23) in the CPT2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CPT2 are known to be pathogenic (PMID: 16781677, 16996287). This variant is present in population databases (rs762366252, gnomAD 0.006%). This variant has not been reported in the literature in individuals affected with CPT2-related conditions. ClinVar contains an entry for this variant (Variation ID: 551107). For these reasons, this variant has been classified as Pathogenic. |
Genome- |
RCV000666078 | SCV004179391 | likely pathogenic | Carnitine palmitoyl transferase II deficiency, severe infantile form | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Baylor Genetics | RCV003472075 | SCV004211060 | likely pathogenic | Encephalopathy, acute, infection-induced, susceptibility to, 4 | 2023-11-17 | criteria provided, single submitter | clinical testing |