Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000671916 | SCV000796950 | likely pathogenic | Hereditary factor XI deficiency disease | 2018-01-05 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000796225 | SCV000935730 | pathogenic | not provided | 2023-08-02 | criteria provided, single submitter | clinical testing | This sequence change falls in intron 10 of the F11 gene. It does not directly change the encoded amino acid sequence of the F11 protein. It affects a nucleotide within the consensus splice site. For these reasons, this variant has been classified as Pathogenic. Variants that disrupt the consensus splice site are a relatively common cause of aberrant splicing (PMID: 17576681, 9536098). Studies have shown that this variant alters mRNA splicing and is expected to lead to the loss of protein expression (PMID: 15946525, 27067486). ClinVar contains an entry for this variant (Variation ID: 555987). This variant has been observed in individuals with Factor XI deficiency (PMID: 15946525, 27067486). It has also been observed to segregate with disease in related individuals. This variant is present in population databases (no rsID available, gnomAD 0.006%). |
Natera, |
RCV001829873 | SCV002082943 | pathogenic | Plasma factor XI deficiency | 2020-11-24 | no assertion criteria provided | clinical testing |