Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000667724 | SCV000792220 | pathogenic | Fanconi anemia complementation group A | 2017-06-12 | criteria provided, single submitter | clinical testing | |
Invitae | RCV002530721 | SCV003442373 | pathogenic | Fanconi anemia | 2022-06-21 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 552462). This premature translational stop signal has been observed in individual(s) with Fanconi anemia (PMID: 17924555, 30792206). This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Trp957*) in the FANCA gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in FANCA are known to be pathogenic (PMID: 19367192). |
Baylor Genetics | RCV000667724 | SCV004196081 | pathogenic | Fanconi anemia complementation group A | 2023-07-09 | criteria provided, single submitter | clinical testing | |
Leiden Open Variation Database | RCV000667724 | SCV001425836 | pathogenic | Fanconi anemia complementation group A | 2020-02-28 | no assertion criteria provided | curation | Curator: Arleen D. Auerbach. Submitter to LOVD: Johan de Winter. |