Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001235195 | SCV001407871 | pathogenic | Glycogen storage disease due to glucose-6-phosphatase deficiency type IA | 2022-05-30 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. This sequence change replaces glycine, which is neutral and non-polar, with arginine, which is basic and polar, at codon 222 of the G6PC protein (p.Gly222Arg). This variant is present in population databases (no rsID available, gnomAD 0.003%). This missense change has been observed in individual(s) with G6PC-related conditions (PMID: 7744838, 7814621, 9359038, 10070617). ClinVar contains an entry for this variant (Variation ID: 961497). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt G6PC protein function. Experimental studies have shown that this missense change affects G6PC function (PMID: 7744838, 7814621, 9359038, 11739393). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. |
Centre for Human Genetics | RCV001235195 | SCV001482272 | pathogenic | Glycogen storage disease due to glucose-6-phosphatase deficiency type IA | 2020-08-20 | criteria provided, single submitter | clinical testing | disease causing |
Natera, |
RCV001235195 | SCV002093319 | pathogenic | Glycogen storage disease due to glucose-6-phosphatase deficiency type IA | 2017-03-17 | no assertion criteria provided | clinical testing |