Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Institute of Human Genetics, |
RCV001261199 | SCV001438069 | likely pathogenic | Maturity-onset diabetes of the young type 2 | 2020-09-21 | criteria provided, single submitter | clinical testing | |
Clinical Genomics, |
RCV002463795 | SCV002605079 | likely risk allele | Maturity onset diabetes mellitus in young | criteria provided, single submitter | research | Potent mutations in GCK gene is associated with poor secretion of insulin. Its associated with milder forms of diabetes, which can be controlled by diet . However, there is no sufficient evidence to ascertain the significance of rs2096271425 in MODY, yet. | |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV001261199 | SCV005077261 | pathogenic | Maturity-onset diabetes of the young type 2 | 2024-04-18 | criteria provided, single submitter | clinical testing | Variant summary: GCK c.1178T>C (p.Met393Thr) results in a non-conservative amino acid change located in the Hexokinase, C-terminal domain (IPR022673) of the encoded protein sequence. Three of five in-silico tools predict a damaging effect of the variant on protein function. The variant was absent in 225042 control chromosomes (gnomAD). c.1178T>C has been reported in the literature in multiple individuals affected with Maturity Onset Diabetes Of The Young 2/Neonatal Diabetes Mellitus (e.g. Osbak_2009, Flanagan_2014, Aglagioglu_2016, Breidbart_2021). These data indicate that the variant is very likely to be associated with disease. The following publications have been ascertained in the context of this evaluation (PMID: 19790256, 26669242, 33852230, 24411943). ClinVar contains an entry for this variant (Variation ID: 981653). Based on the evidence outlined above, the variant was classified as pathogenic. |