Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001261548 | SCV003785524 | pathogenic | Mitochondrial trifunctional protein deficiency | 2024-01-02 | criteria provided, single submitter | clinical testing | This sequence change affects an acceptor splice site in intron 5 of the HADHB gene. It is expected to disrupt RNA splicing. Variants that disrupt the donor or acceptor splice site typically lead to a loss of protein function (PMID: 16199547), and loss-of-function variants in HADHB are known to be pathogenic (PMID: 9259266, 12754706). This variant is present in population databases (rs112842641, gnomAD 0.008%). Disruption of this splice site has been observed in individual(s) with mitochondrial trifunctional protein deficiency (PMID: 35433169). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 916614). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. For these reasons, this variant has been classified as Pathogenic. |
Department of Medical Genetics, |
RCV001261548 | SCV001244851 | uncertain significance | Mitochondrial trifunctional protein deficiency | 2020-04-24 | no assertion criteria provided | clinical testing |