Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001298493 | SCV001487551 | benign | Long QT syndrome | 2022-12-06 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001567242 | SCV001790893 | likely benign | not provided | 2015-03-03 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002380002 | SCV002694882 | uncertain significance | Cardiovascular phenotype | 2022-08-24 | criteria provided, single submitter | clinical testing | The p.E450K variant (also known as c.1348G>A), located in coding exon 10 of the KCNQ1 gene, results from a G to A substitution at nucleotide position 1348. The glutamic acid at codon 450 is replaced by lysine, an amino acid with similar properties. This alteration has been reported in a sudden unexplained death cohort; however, clinical details were limited (Huang J et al. J Forensic Sci, 2015 Mar;60:351-6). This amino acid position is not well conserved in available vertebrate species. In addition, the in silico prediction for this alteration is inconclusive. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
All of Us Research Program, |
RCV001298493 | SCV004826565 | uncertain significance | Long QT syndrome | 2023-08-15 | criteria provided, single submitter | clinical testing | This missense variant replaces glutamic acid with lysine at codon 450 of the KCNQ1 protein. Computational prediction is inconclusive regarding the impact of this variant on protein structure and function (internally defined REVEL score threshold 0.5 < inconclusive < 0.7, PMID: 27666373). To our knowledge, functional studies have not been reported for this variant. This variant has not been reported in individuals affected with KCNQ1-related disorders in the literature. This variant has been identified in 2/250152 chromosomes in the general population by the Genome Aggregation Database (gnomAD). The available evidence is insufficient to determine the role of this variant in disease conclusively. Therefore, this variant is classified as a Variant of Uncertain Significance. |