Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV000617855 | SCV000738251 | pathogenic | Cardiovascular phenotype | 2017-12-12 | criteria provided, single submitter | clinical testing | The c.1514+1G>A intronic pathogenic mutation results from a G to A substitution one nucleotide after coding exon 11 of the KCNQ1 gene, and is located in the C-terminal domain. This alteration has been reported in a long QT syndrome (LQTS) cohort (Napolitano C et al. JAMA, 2005 Dec;294:2975-80). In addition to the clinical data presented in the literature, alterations that disrupt the canonical splice site are expected to cause aberrant splicing, resulting in an abnormal protein or a transcript that is subject to nonsense-mediated mRNA decay. As such, this alteration is classified as a disease-causing mutation. |
Clin |
RCV000577184 | SCV000678867 | not provided | Long QT syndrome 1 | no assertion provided | literature only |