Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000636848 | SCV000758289 | uncertain significance | Autosomal recessive limb-girdle muscular dystrophy type 2C | 2022-09-01 | criteria provided, single submitter | clinical testing | This sequence change replaces threonine, which is neutral and polar, with alanine, which is neutral and non-polar, at codon 8 of the SGCG protein (p.Thr8Ala). This variant is present in population databases (rs771657671, gnomAD 0.04%). This variant has not been reported in the literature in individuals affected with SGCG-related conditions. ClinVar contains an entry for this variant (Variation ID: 530808). Algorithms developed to predict the effect of missense changes on protein structure and function output the following: SIFT: "Tolerated"; PolyPhen-2: "Benign"; Align-GVGD: "Class C0". The alanine amino acid residue is found in multiple mammalian species, which suggests that this missense change does not adversely affect protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV004025483 | SCV004947555 | uncertain significance | Inborn genetic diseases | 2021-08-28 | criteria provided, single submitter | clinical testing | The c.22A>G (p.T8A) alteration is located in exon 2 (coding exon 1) of the SGCG gene. This alteration results from a A to G substitution at nucleotide position 22, causing the threonine (T) at amino acid position 8 to be replaced by an alanine (A). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Natera, |
RCV000636848 | SCV002086083 | uncertain significance | Autosomal recessive limb-girdle muscular dystrophy type 2C | 2019-11-11 | no assertion criteria provided | clinical testing |