Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV002289903 | SCV000729237 | uncertain significance | not provided | 2022-10-05 | criteria provided, single submitter | clinical testing | Published functional studies suggest a damaging effect, as the c.243+6>A variant disrupts the natural donor splice site of exon 2 and activates a cryptic splice site in intron 2, resulting in a frameshift due to an inclusion of 10 base pairs of intron 2 (Xie et al., 2022); In silico analysis supports a deleterious effect on splicing; This variant is associated with the following publications: (PMID: 35813381) |
Labcorp Genetics |
RCV000874699 | SCV001016910 | benign | Autosomal recessive limb-girdle muscular dystrophy type 2E | 2024-11-22 | criteria provided, single submitter | clinical testing | |
Illumina Laboratory Services, |
RCV001148511 | SCV001309410 | benign | Qualitative or quantitative defects of beta-sarcoglycan | 2017-04-27 | criteria provided, single submitter | clinical testing | This variant was observed as part of a predisposition screen in an ostensibly healthy population. A literature search was performed for the gene, cDNA change, and amino acid change (where applicable). No publications were found based on this search. Allele frequency data from public databases was too high to be consistent with this variant causing disease. Therefore, this variant is classified as benign. |
Breakthrough Genomics, |
RCV002289903 | SCV005257341 | likely benign | not provided | criteria provided, single submitter | not provided |