Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000009256 | SCV000797524 | likely pathogenic | Autosomal recessive limb-girdle muscular dystrophy type 2E | 2018-02-05 | criteria provided, single submitter | clinical testing | |
Mendelics | RCV000009256 | SCV001136738 | pathogenic | Autosomal recessive limb-girdle muscular dystrophy type 2E | 2019-05-28 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000009256 | SCV002261967 | uncertain significance | Autosomal recessive limb-girdle muscular dystrophy type 2E | 2022-08-21 | criteria provided, single submitter | clinical testing | This sequence change replaces methionine, which is neutral and non-polar, with lysine, which is basic and polar, at codon 100 of the SGCB protein (p.Met100Lys). This variant is not present in population databases (gnomAD no frequency). This missense change has been observed in individual(s) with SGCB-related conditions (PMID: 8968749, 12566530, 15938574). ClinVar contains an entry for this variant (Variation ID: 8718). Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be disruptive. Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Baylor Genetics | RCV000009256 | SCV004208743 | likely pathogenic | Autosomal recessive limb-girdle muscular dystrophy type 2E | 2023-08-28 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000009256 | SCV000029474 | pathogenic | Autosomal recessive limb-girdle muscular dystrophy type 2E | 1996-12-01 | no assertion criteria provided | literature only |