Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000539411 | SCV000627466 | likely benign | Long QT syndrome | 2023-03-10 | criteria provided, single submitter | clinical testing | |
Gene |
RCV000829311 | SCV000971027 | likely benign | not provided | 2018-06-14 | criteria provided, single submitter | clinical testing | This variant is considered likely benign or benign based on one or more of the following criteria: it is a conservative change, it occurs at a poorly conserved position in the protein, it is predicted to be benign by multiple in silico algorithms, and/or has population frequency not consistent with disease. |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV001729623 | SCV006074382 | likely benign | not specified | 2025-04-24 | criteria provided, single submitter | clinical testing | Variant summary: KCNH2 c.2692+7C>G alters a non-conserved nucleotide located at a position not widely known to affect splicing. Consensus agreement among computation tools predict no significant impact on normal splicing. However, these predictions have yet to be confirmed by functional studies. The frequency data for this variant in gnomAD is considered unreliable, as metrics indicate poor data quality at this position. To our knowledge, no occurrence of c.2692+7C>G in individuals affected with KCNH2-related conditions and no experimental evidence demonstrating its impact on protein function have been reported. ClinVar contains an entry for this variant (Variation ID: 456915). Based on the evidence outlined above, the variant was classified as likely benign. |
Clinical Genetics, |
RCV001729623 | SCV001979267 | benign | not specified | no assertion criteria provided | clinical testing | ||
Joint Genome Diagnostic Labs from Nijmegen and Maastricht, |
RCV001729623 | SCV001979767 | benign | not specified | no assertion criteria provided | clinical testing |