Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001280297 | SCV003312231 | uncertain significance | MHC class II deficiency | 2022-02-06 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be tolerated. ClinVar contains an entry for this variant (Variation ID: 991991). This variant has not been reported in the literature in individuals affected with CIITA-related conditions. This variant is present in population databases (rs765687423, gnomAD 0.007%). This sequence change replaces proline, which is neutral and non-polar, with glutamine, which is neutral and polar, at codon 517 of the CIITA protein (p.Pro517Gln). |
Ambry Genetics | RCV003263919 | SCV003956598 | uncertain significance | Inborn genetic diseases | 2023-04-19 | criteria provided, single submitter | clinical testing | The c.1550C>A (p.P517Q) alteration is located in exon 11 (coding exon 11) of the CIITA gene. This alteration results from a C to A substitution at nucleotide position 1550, causing the proline (P) at amino acid position 517 to be replaced by a glutamine (Q). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Natera, |
RCV001280297 | SCV001467463 | uncertain significance | MHC class II deficiency | 2020-04-24 | no assertion criteria provided | clinical testing |