Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV000491259 | SCV000580431 | pathogenic | Hereditary cancer-predisposing syndrome | 2020-11-14 | criteria provided, single submitter | clinical testing | The c.790dupC pathogenic mutation, located in coding exon 4 of the MSH2 gene, results from a duplication of C at nucleotide position 790, causing a translational frameshift with a predicted alternate stop codon (p.Q264Pfs*20). This variant was not reported in population-based cohorts in the Genome Aggregation Database (gnomAD). This alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay. As such, this alteration is interpreted as a disease-causing mutation. |
Labcorp Genetics |
RCV003593971 | SCV004292541 | pathogenic | Hereditary nonpolyposis colorectal neoplasms | 2023-12-22 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gln264Profs*20) in the MSH2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in MSH2 are known to be pathogenic (PMID: 15849733, 24362816). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with MSH2-related conditions. ClinVar contains an entry for this variant (Variation ID: 428460). For these reasons, this variant has been classified as Pathogenic. |