Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Institute of Medical Genetics and Applied Genomics, |
RCV001543470 | SCV001762056 | pathogenic | not provided | 2021-06-17 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000781954 | SCV004298751 | pathogenic | Severe X-linked myotubular myopathy | 2023-09-30 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. ClinVar contains an entry for this variant (Variation ID: 633470). This premature translational stop signal has been observed in individual(s) with centronuclear myopathy (PMID: 25957634). This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Gln461*) in the MTM1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in MTM1 are known to be pathogenic (PMID: 9305655, 10063835). |
Clinical Molecular Genetics Laboratory, |
RCV000781954 | SCV000920399 | pathogenic | Severe X-linked myotubular myopathy | 2007-08-02 | no assertion criteria provided | clinical testing |