Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000732987 | SCV000860994 | pathogenic | not provided | 2018-04-23 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003768229 | SCV004651050 | pathogenic | Methylcobalamin deficiency type cblG | 2023-12-26 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Ile1201Leufs*3) in the MTR gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in MTR are known to be pathogenic (PMID: 9683607, 12068375). This variant is present in population databases (no rsID available, gnomAD 0.0009%). This premature translational stop signal has been observed in individual(s) with MRT-related conditions (PMID: 34269512). ClinVar contains an entry for this variant (Variation ID: 596996). For these reasons, this variant has been classified as Pathogenic. |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV003768229 | SCV005203786 | pathogenic | Methylcobalamin deficiency type cblG | 2024-07-02 | criteria provided, single submitter | clinical testing | Variant summary: MTR c.3600delC (p.Ile1201LeufsX3) results in a premature termination codon, predicted to cause a truncation of the encoded protein or absence of the protein due to nonsense mediated decay, which are commonly known mechanisms for disease. The variant allele was found at a frequency of 4e-06 in 251422 control chromosomes. c.3600delC has been reported in the literature as a compound heterozygous genotype in at-least one individual affected with isolated homocysteine re-methylation defect, a feature of Methylcobalamin deficiency type cblG (example, Whitehouse_2023). The following publication has been ascertained in the context of this evaluation (PMID: 37404677). ClinVar contains an entry for this variant (Variation ID: 596996). Based on the evidence outlined above, the variant was classified as pathogenic. |