Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000523352 | SCV000620904 | uncertain significance | not provided | 2017-09-15 | criteria provided, single submitter | clinical testing | A variant of uncertain significance has been identified in the MTR gene. The P301A variant has not been published as a pathogenic variant, nor has it been reported as a benign variant to our knowledge. The P301A variant is observed in 7/66738 (0.01%) alleles from individuals of non-Finnish Eropean background (Lek et al., 2016; 1000 Genomes Consortium et al., 2015; Exome Variant Server). The P301A variant is a semi-conservative amino acid substitution, which may impact secondary protein structure as these residues differ in some properties. This substitution occurs at a position that is conserved across species. In silico analysis predicts this variant is probably damaging to the protein structure/function. Based on the currently available information, it is unclear whether this variant is a pathogenic variant or a rare benign variant. |
Labcorp Genetics |
RCV001858021 | SCV002264765 | uncertain significance | Methylcobalamin deficiency type cblG | 2022-03-12 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with alanine, which is neutral and non-polar, at codon 301 of the MTR protein (p.Pro301Ala). This variant is present in population databases (rs766371534, gnomAD 0.009%). This variant has not been reported in the literature in individuals affected with MTR-related conditions. ClinVar contains an entry for this variant (Variation ID: 452126). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Deleterious"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0"). In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |