Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000421377 | SCV000513834 | uncertain significance | not provided | 2024-12-30 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 21738395, 16963483, 20613545) |
Counsyl | RCV000665055 | SCV000789114 | uncertain significance | Autosomal recessive nonsyndromic hearing loss 2; Usher syndrome type 1 | 2017-01-10 | criteria provided, single submitter | clinical testing | |
Laboratory for Molecular Medicine, |
RCV000825981 | SCV000967469 | uncertain significance | not specified | 2020-02-28 | criteria provided, single submitter | clinical testing | The p.Ala1340Thr variant in MYO7A has been previously reported in at least 3 individuals with hearing loss or Usher syndrome, however a variant affecting the remaining copy of MYO7A was not identified (Cremers 2007, Kimberling 2010, Vozzi 2011, LMM Unpublished data). This variant has also been identified in 0.13% (14/10342) of Ashkenazi Jewish chromosomes by gnomAD (http://gnomad.broadinstitute.org). Computational prediction tools and conservation analysis do not provide strong support for or against an impact to the protein. In summary, the clinical significance of this variant is uncertain. ACMG/AMP Criteria applied: BS1_Supporting. |
Labcorp Genetics |
RCV000421377 | SCV001541607 | likely benign | not provided | 2024-10-30 | criteria provided, single submitter | clinical testing | |
Natera, |
RCV001273495 | SCV001456606 | uncertain significance | Usher syndrome type 1B | 2020-09-16 | no assertion criteria provided | clinical testing | |
Prevention |
RCV004735507 | SCV005362040 | uncertain significance | MYO7A-related disorder | 2024-09-26 | no assertion criteria provided | clinical testing | The MYO7A c.4018G>A variant is predicted to result in the amino acid substitution p.Ala1340Thr. This variant has been reported in the heterozygous state in three individuals with deafness or Usher syndrome, although one of these individuals was also heterozygous for a truncating variant in another Usher syndrome related gene (Kimberling et al. 2010. PubMed ID: 20613545; Vozzi et al. 2011. PubMed ID: 21738395; Cremers et al. 2006. PubMed ID: 16963483). This variant is reported in 0.14% of alleles in individuals of Ashkenazi Jewish descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |