Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002512914 | SCV003439382 | pathogenic | Peroxisome biogenesis disorder | 2023-11-25 | criteria provided, single submitter | clinical testing | This sequence change affects a donor splice site in intron 7 of the PEX6 gene. RNA analysis indicates that disruption of this splice site induces altered splicing and may result in an absent or disrupted protein product. This variant is not present in population databases (gnomAD no frequency). Disruption of this splice site has been observed in individual(s) with PEX6-related conditions (PMID: 10408779, 19877282). ClinVar contains an entry for this variant (Variation ID: 8123). Studies have shown that disruption of this splice site results in skipping of either exon 7 or both exons 6 and 7 and introduces a premature termination codon (PMID: 10408779). The resulting mRNA is expected to undergo nonsense-mediated decay. For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV003473057 | SCV004201586 | pathogenic | Heimler syndrome 2 | 2023-02-26 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000008594 | SCV000028802 | pathogenic | Peroxisome biogenesis disorder 4A (Zellweger) | 1999-01-01 | no assertion criteria provided | literature only |