Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Leeds Amelogenesis Imperfecta Research Group, |
RCV000240712 | SCV000264801 | pathogenic | Heimler syndrome 2 | 2015-10-01 | criteria provided, single submitter | research | Newly identified |
Counsyl | RCV000675085 | SCV000800593 | uncertain significance | Peroxisome biogenesis disorder 4A (Zellweger); Peroxisome biogenesis disorder 4B | 2017-09-15 | criteria provided, single submitter | clinical testing | |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV004689680 | SCV005185244 | uncertain significance | not specified | 2024-05-20 | criteria provided, single submitter | clinical testing | Variant summary: PEX6 c.654C>G (p.Phe218Leu) results in a non-conservative amino acid change in the encoded protein sequence. Four of five in-silico tools predict a damaging effect of the variant on protein function. The variant allele was found at a frequency of 4e-06 in 251178 control chromosomes. The available data on variant occurrences in the general population are insufficient to allow any conclusion about variant significance. c.654C>G has been reported in the literature in at-least one individual affected with Heimler syndrome (example: Smith_2016) . These data do not allow any conclusion about variant significance. To our knowledge, no experimental evidence demonstrating an impact on protein function has been reported. The following publication has been ascertained in the context of this evaluation (PMID: 27302843). ClinVar contains an entry for this variant (Variation ID: 224318). Based on the evidence outlined above, the variant was classified as uncertain significance. |