Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000148747 | SCV002258134 | uncertain significance | Thrombophilia due to protein C deficiency, autosomal dominant | 2021-08-31 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Tolerated"; PolyPhen-2: "Possibly Damaging"; Align-GVGD: "Class C0"). ClinVar contains an entry for this variant (Variation ID: 161341). This variant is also known as p.Gly72Arg. This missense change has been observed in individual(s) with protein C deficiency (PMID: 7792728). This variant is present in population databases (rs374476971, ExAC 0.005%). This sequence change replaces glycine with arginine at codon 114 of the PROC protein (p.Gly114Arg). The glycine residue is moderately conserved and there is a moderate physicochemical difference between glycine and arginine. |
Fulgent Genetics, |
RCV002478414 | SCV002787224 | uncertain significance | Thrombophilia due to protein C deficiency, autosomal recessive; Thrombophilia due to protein C deficiency, autosomal dominant | 2021-07-07 | criteria provided, single submitter | clinical testing | |
CSER _CC_NCGL, |
RCV000148747 | SCV000190484 | uncertain significance | Thrombophilia due to protein C deficiency, autosomal dominant | 2014-06-01 | no assertion criteria provided | research | |
ISTH- |
RCV002243820 | SCV002515555 | uncertain significance | Reduced protein C activity | no assertion criteria provided | clinical testing |