Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001358958 | SCV001554815 | uncertain significance | not provided | 2022-03-21 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Deleterious"; PolyPhen-2: "Benign"; Align-GVGD: "Class C55"). ClinVar contains an entry for this variant (Variation ID: 1050981). This variant has not been reported in the literature in individuals affected with PTH1R-related conditions. This variant is present in population databases (rs200667470, gnomAD 0.2%). This sequence change replaces glutamic acid, which is acidic and polar, with lysine, which is basic and polar, at codon 549 of the PTH1R protein (p.Glu549Lys). |
Fulgent Genetics, |
RCV002504587 | SCV002814611 | uncertain significance | Primary failure of tooth eruption; Chondrodysplasia Blomstrand type; Eiken syndrome; Metaphyseal chondrodysplasia, Jansen type | 2021-08-19 | criteria provided, single submitter | clinical testing | |
3billion | RCV004731135 | SCV005328681 | likely benign | Chondrodysplasia Blomstrand type; Eiken syndrome | 2024-09-20 | criteria provided, single submitter | clinical testing | The homozygous variant was found in patients diagnosed with another variant in a different gene, with no symptoms related to the gene containing the homozygous variant. |