Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Center for Genomic Medicine, |
RCV000171395 | SCV000221592 | likely pathogenic | not provided | criteria provided, single submitter | research | ||
Blueprint Genetics | RCV001073434 | SCV001238975 | pathogenic | Retinal dystrophy | 2019-02-12 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002517650 | SCV003439429 | pathogenic | PRPH2-related disorder | 2022-02-20 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt PRPH2 protein function. ClinVar contains an entry for this variant (Variation ID: 191208). This missense change has been observed in individuals with PRPH2-related conditions (PMID: 26061163, 26355662; Invitae). This variant is not present in population databases (gnomAD no frequency). This sequence change replaces cysteine, which is neutral and slightly polar, with tyrosine, which is neutral and polar, at codon 166 of the PRPH2 protein (p.Cys166Tyr). |
Leiden Open Variation Database | RCV000171395 | SCV001744953 | uncertain significance | not provided | 2021-04-29 | no assertion criteria provided | curation | Curator: Global Variome, with Curator vacancy. Submitter to LOVD: Manon Peeters. |