Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002513030 | SCV003443028 | pathogenic | not provided | 2023-07-29 | criteria provided, single submitter | clinical testing | ClinVar contains an entry for this variant (Variation ID: 13099). For these reasons, this variant has been classified as Pathogenic. Variants that disrupt the consensus splice site are a relatively common cause of aberrant splicing (PMID: 17576681, 9536098). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. This variant is also known as 324G->A. This variant has been observed in individuals with rod-cone dystrophy (PMID: 11868161). It has also been observed to segregate with disease in related individuals. This variant is present in population databases (rs766278489, gnomAD 0.002%). This sequence change affects codon 47 of the RLBP1 mRNA. It is a 'silent' change, meaning that it does not change the encoded amino acid sequence of the RLBP1 protein. This variant also falls at the last nucleotide of exon 4, which is part of the consensus splice site for this exon. |
OMIM | RCV000013976 | SCV000034223 | pathogenic | Newfoundland cone-rod dystrophy | 2002-04-01 | no assertion criteria provided | literature only | |
OMIM | RCV000013977 | SCV000034224 | pathogenic | Retinitis punctata albescens | 2002-04-01 | no assertion criteria provided | literature only |