Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000559969 | SCV000658539 | uncertain significance | Hyperkalemic periodic paralysis | 2024-01-15 | criteria provided, single submitter | clinical testing | This sequence change replaces lysine, which is basic and polar, with glutamine, which is neutral and polar, at codon 888 of the SCN4A protein (p.Lys888Gln). This variant is present in population databases (rs202155883, gnomAD 0.005%). This variant has not been reported in the literature in individuals affected with SCN4A-related conditions. ClinVar contains an entry for this variant (Variation ID: 477407). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is not expected to disrupt SCN4A protein function with a negative predictive value of 95%. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Fulgent Genetics, |
RCV005004245 | SCV002796977 | uncertain significance | Potassium-aggravated myotonia; Paramyotonia congenita of Von Eulenburg; Hypokalemic periodic paralysis, type 2; Hyperkalemic periodic paralysis; Congenital myasthenic syndrome 16; Congenital myopathy 22A, classic; Congenital myopathy 22B, severe fetal | 2024-05-09 | criteria provided, single submitter | clinical testing | |
Revvity Omics, |
RCV003139862 | SCV003821276 | uncertain significance | not provided | 2022-09-08 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004024398 | SCV004944257 | uncertain significance | Inborn genetic diseases | 2023-12-12 | criteria provided, single submitter | clinical testing | The c.2662A>C (p.K888Q) alteration is located in exon 14 (coding exon 14) of the SCN4A gene. This alteration results from a A to C substitution at nucleotide position 2662, causing the lysine (K) at amino acid position 888 to be replaced by a glutamine (Q). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |