Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001247448 | SCV001420871 | likely pathogenic | not provided | 2019-11-16 | criteria provided, single submitter | clinical testing | In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. This variant has been reported to affect SLC4A1 protein function (PMID: 28045035, 22518001). This variant has been observed in individual(s) with autosomal dominant renal tubular acidosis or nephrocalcinosis (PMID: 22518001, Invitae). It has also been observed to segregate with disease in related individuals. This variant is not present in population databases (ExAC no frequency). This sequence change replaces methionine with threonine at codon 909 of the SLC4A1 protein (p.Met909Thr). The methionine residue is moderately conserved and there is a moderate physicochemical difference between methionine and threonine. |
Fulgent Genetics, |
RCV002499429 | SCV002811530 | likely pathogenic | BLOOD GROUP--SWANN SYSTEM; BLOOD GROUP--WALDNER TYPE; BLOOD GROUP--FROESE; BLOOD GROUP--WRIGHT ANTIGEN; Southeast Asian ovalocytosis; Hereditary spherocytosis type 4; BLOOD GROUP--DIEGO SYSTEM; Cryohydrocytosis; Autosomal dominant distal renal tubular acidosis; Renal tubular acidosis, distal, 4, with hemolytic anemia; Malaria, susceptibility to | 2021-09-20 | criteria provided, single submitter | clinical testing |