Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001052785 | SCV001217011 | pathogenic | not provided | 2022-09-27 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Trp499*) in the ABCA4 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in ABCA4 are known to be pathogenic (PMID: 10958761, 24938718, 25312043, 26780318). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with Stargardt disease (PMID: 28512305). ClinVar contains an entry for this variant (Variation ID: 848932). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. For these reasons, this variant has been classified as Pathogenic. |
Blueprint Genetics | RCV001075839 | SCV001241478 | pathogenic | Retinal dystrophy | 2019-08-01 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001052785 | SCV002064151 | pathogenic | not provided | 2022-01-24 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Identified in an individual with Stargardt disease in published literature (Huang et al., 2017); This variant is associated with the following publications: (PMID: 33301772, 28512305) |
Fulgent Genetics, |
RCV005036344 | SCV005663571 | likely pathogenic | Cone-rod dystrophy 3; Age related macular degeneration 2; Severe early-childhood-onset retinal dystrophy; Retinitis pigmentosa 19 | 2024-05-30 | criteria provided, single submitter | clinical testing |