Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000760329 | SCV000890185 | pathogenic | not provided | 2023-05-15 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |
Institute of Human Genetics, |
RCV001253033 | SCV001428550 | likely pathogenic | Tuberous sclerosis 1 | 2018-05-08 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV001253033 | SCV002040653 | pathogenic | Tuberous sclerosis 1 | 2021-11-07 | criteria provided, single submitter | clinical testing | |
Division of Genomic Medicine, |
RCV001253033 | SCV002549093 | pathogenic | Tuberous sclerosis 1 | 2022-07-17 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001253033 | SCV003238527 | pathogenic | Tuberous sclerosis 1 | 2024-11-04 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gln936*) in the TSC1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in TSC1 are known to be pathogenic (PMID: 10227394, 17304050). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with tuberous sclerosis complex (PMID: 36232477). ClinVar contains an entry for this variant (Variation ID: 49002). For these reasons, this variant has been classified as Pathogenic. |
Tuberous sclerosis database |
RCV000042253 | SCV000066040 | not provided | Tuberous sclerosis syndrome | no assertion provided | curation |