Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ce |
RCV001531120 | SCV001746096 | pathogenic | not provided | 2021-04-01 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV001587463 | SCV001821956 | pathogenic | Tyrosinase-negative oculocutaneous albinism | 2021-07-22 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001531120 | SCV002294667 | pathogenic | not provided | 2023-01-01 | criteria provided, single submitter | clinical testing | This sequence change affects a donor splice site in intron 2 of the TYR gene. It is expected to disrupt RNA splicing. Variants that disrupt the donor or acceptor splice site typically lead to a loss of protein function (PMID: 16199547), and loss-of-function variants in TYR are known to be pathogenic (PMID: 23504663). This variant is present in population databases (rs763715899, gnomAD 0.003%). Disruption of this splice site has been observed in individual(s) with oculocutaneous albinism (PMID: 13680365, 32115698). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 1175803). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV003463045 | SCV004207619 | likely pathogenic | SKIN/HAIR/EYE PIGMENTATION 3, LIGHT/DARK SKIN | 2023-03-07 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV005005950 | SCV005631367 | likely pathogenic | Tyrosinase-negative oculocutaneous albinism; Oculocutaneous albinism type 1B; SKIN/HAIR/EYE PIGMENTATION 3, LIGHT/DARK SKIN | 2024-04-08 | criteria provided, single submitter | clinical testing |