Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Athena Diagnostics | RCV000710494 | SCV000840728 | likely pathogenic | not provided | 2018-08-03 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001045056 | SCV001208885 | pathogenic | Polyglandular autoimmune syndrome, type 1 | 2022-12-20 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 585381). This variant has not been reported in the literature in individuals affected with AIRE-related conditions. This variant is present in population databases (rs763695515, gnomAD 0.003%). This sequence change creates a premature translational stop signal (p.Arg433Alafs*71) in the AIRE gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in AIRE are known to be pathogenic (PMID: 11524731, 26141571). |
Gene |
RCV000710494 | SCV002762029 | likely pathogenic | not provided | 2022-06-08 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |
Fulgent Genetics, |
RCV001045056 | SCV002786826 | likely pathogenic | Polyglandular autoimmune syndrome, type 1 | 2022-02-07 | criteria provided, single submitter | clinical testing |