Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000666118 | SCV000790361 | likely pathogenic | Polyglandular autoimmune syndrome, type 1 | 2017-03-16 | criteria provided, single submitter | clinical testing | |
NIHR Bioresource Rare Diseases, |
RCV001027543 | SCV001190113 | pathogenic | Inherited Immunodeficiency Diseases | 2019-01-01 | criteria provided, single submitter | research | |
Labcorp Genetics |
RCV000666118 | SCV001591354 | pathogenic | Polyglandular autoimmune syndrome, type 1 | 2021-05-21 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Disruption of the initiator codon has been observed in individual(s) with autosomal recessive autoimmune polyendocrinopathy syndrome (PMID: 19758376, 28446514, 28911151,16965330). In at least one individual the data is consistent with the variant being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 551136). While this variant is present in population databases (rs121434258), the frequency information is unreliable, as metrics indicate poor data quality at this position in the ExAC database. This sequence change affects the initiator methionine of the AIRE mRNA. The next in-frame methionine is located at codon 184. |