Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001242405 | SCV001415491 | uncertain significance | Polyglandular autoimmune syndrome, type 1 | 2022-08-22 | criteria provided, single submitter | clinical testing | This sequence change replaces serine, which is neutral and polar, with cysteine, which is neutral and slightly polar, at codon 250 of the AIRE protein (p.Ser250Cys). This variant is present in population databases (rs141480813, gnomAD 0.05%). This missense change has been observed in individual(s) with autoimmunity and immunodeficiency (PMID: 25068407). ClinVar contains an entry for this variant (Variation ID: 967483). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Deleterious"; PolyPhen-2: "Possibly Damaging"; Align-GVGD: "Class C0"). In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Fulgent Genetics, |
RCV001242405 | SCV002782400 | uncertain significance | Polyglandular autoimmune syndrome, type 1 | 2021-08-03 | criteria provided, single submitter | clinical testing | |
Natera, |
RCV001242405 | SCV002083872 | uncertain significance | Polyglandular autoimmune syndrome, type 1 | 2020-03-17 | no assertion criteria provided | clinical testing | |
Prevention |
RCV004754713 | SCV005354014 | uncertain significance | AIRE-related disorder | 2024-08-08 | no assertion criteria provided | clinical testing | The AIRE c.748A>T variant is predicted to result in the amino acid substitution p.Ser250Cys. This variant was reported in an individual with autoimmunity and immunodeficiency (Bellacchio et al. 2014. PubMed ID: 25068407; Fierabracci et al. 2022. PubMed ID: 35683627). This variant is reported in 0.041% of alleles in individuals of African descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |