Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001859123 | SCV002188285 | likely benign | Hypercholesterolemia, autosomal dominant, type B; Familial hypobetalipoproteinemia 1 | 2025-01-15 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002402548 | SCV002708774 | uncertain significance | Cardiovascular phenotype | 2023-12-15 | criteria provided, single submitter | clinical testing | The p.I523T variant (also known as c.1568T>C), located in coding exon 12 of the APOB gene, results from a T to C substitution at nucleotide position 1568. The isoleucine at codon 523 is replaced by threonine, an amino acid with similar properties. This amino acid position is not well conserved in available vertebrate species. In addition, this alteration is predicted to be deleterious by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Fulgent Genetics, |
RCV001859123 | SCV002785910 | uncertain significance | Hypercholesterolemia, autosomal dominant, type B; Familial hypobetalipoproteinemia 1 | 2021-10-12 | criteria provided, single submitter | clinical testing | |
Gene |
RCV003332299 | SCV004039643 | uncertain significance | not provided | 2023-04-05 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |