Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001773430 | SCV001993417 | uncertain significance | not provided | 2023-09-15 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; In silico analysis supports that this missense variant does not alter protein structure/function |
Labcorp Genetics |
RCV001876068 | SCV002114136 | likely benign | Hypercholesterolemia, autosomal dominant, type B; Familial hypobetalipoproteinemia 1 | 2024-10-18 | criteria provided, single submitter | clinical testing | |
MGZ Medical Genetics Center | RCV002290619 | SCV002580914 | uncertain significance | Hypercholesterolemia, autosomal dominant, type B | 2022-07-12 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002429817 | SCV002681199 | uncertain significance | Cardiovascular phenotype | 2022-01-10 | criteria provided, single submitter | clinical testing | The c.8198T>C (p.V2733A) alteration is located in exon 26 (coding exon 26) of the APOB gene. This alteration results from a T to C substitution at nucleotide position 8198, causing the valine (V) at amino acid position 2733 to be replaced by an alanine (A). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Fulgent Genetics, |
RCV001876068 | SCV002790491 | uncertain significance | Hypercholesterolemia, autosomal dominant, type B; Familial hypobetalipoproteinemia 1 | 2021-09-28 | criteria provided, single submitter | clinical testing |